NEW RESEARCH: Coronavirus Has A Second Route For Attack!

I can bet that this highly-incovenient research will not be acknowledged

by the NZ health authorities or Bill Gates or Dr Fauchi who are pushing 

for a vaccine

Coronavirus could attack 

immune system like HIV by 

targeting protective cells, 

warn scientists

- Researchers in China and the US find that the virus 
that causes Covid-19 can destroy the T cells that are 
supposed to protect the body from harmful invaders
- One doctor said concern is growing in medical circles 
that effect could be similar to HIV

SCMP,

12 April, 2020

The coronavirus that causes Covid-19 could kill the powerful immune cells that are 

supposed to kill the virus instead, scientists have warned.

The surprise discovery, made by a team of researchers from Shanghai and New York, 

coincided with frontline doctors’ observation that Covid-19 could attack the human 

immune system and cause damage similar to that found in HIV patients.

Lu Lu, from Fudan University in Shanghai, and Jang Shibo, from the New York Blood 

Centre, joined the living virus, which is officially known as Sars-CoV-2, to 

laboratory-grown T lymphocyte cell lines.

T lymphocytes, also known as T cells, play a central role in identifying and eliminating 

alien invaders in the body.

They do this by capturing a cell infected by a virus, boring a hole in its membrane and 

injecting toxic chemicals into the cell. These chemicals then kill both the virus and 

infected cell and tear them to pieces.

The researchers did the same experiment with severe acute respiratory syndrome, or 

Sars, another coronavirus, and found that the Sars virus did not have the ability to 

infect T cells.

The reason, they suspected, was the lack of a membrane fusion function. Sars, which 

killed hundreds in a 2003 outbreak, can only infect cells carrying a specific receptor 

protein known as ACE2, and this protein has an extremely low presence in T cells.

Further investigations into the coronavirus infection on primary T cells would evoke 

“new ideas about pathogenic mechanisms and therapeutic interventions”, the 

researchers said in a paper published in the peer-reviewed journal Cellular & 

Molecular Immunology this week.

A doctor who works in a public hospital treating Covid-19 patients in Beijing said the 

discovery added another piece of evidence to a growing concern in medical circles 

that the coronavirus could sometimes behave like some of the most notorious viruses 

that directly attack the human immune system.

“More and more people compare it to HIV,” said the doctor who requested not to be 

named due to the sensitivity of the issue.

In February, Chen Yongwen and his colleagues at the PLA’s Institute of Immunology 

released a clinical report warning that the number of T cells could drop significantly 
in Covid-19 patients, especially when they were elderly or required treatment in 
intensive care units. The lower the T cell count, the higher the risk of death.

Doctors who had seen the bodies said the damage to the internal organs was similar 

to a combination of Sars and Aids.

The gene behind the fusion function in Sars-CoV-2 was not found in other 

coronaviruses in human or animals.

But some deadly human viruses such as Aids and Ebola have similar sequences, 

prompting speculation that the novel coronavirus might have been spreading quietly 

in human societies for a long time before causing this pandemic.

t there was one major difference between Sars-CoV-2 and HIV, according to the new 

study.

HIV can replicate in the T cells and turn them into factories to generate more copies 

to infect other cells.

But Lu and Jiang did not observe any growth of the coronavirus after it entered the 

T-cells, suggesting that the virus and T-cells might end up dying together.

The study gives rise to some new questions. For instance, the coronavirus can exist 
for weeks on some patients without causing any symptoms. How it interacted with 
the T cells in these patients remained unclear.

Some critically ill patients also experienced cytokine storms, where the immune 

system overreacts and attacks healthy cells.

But why and how the coronavirus triggers that effect remains poorly understood