You’ve
got one group of people saying die-offs of marine mammals is due to
Fukushima radiation and has got nothing to do with global warming.
You’ve got another saying it’s due to the consequences of a
warming Pacific (such as algal blooms) and ne'er the twain shall meet apparently.
This
is absolutely ludicrous.
Both
sides are correct and what we have is a perfect storm of factors
coming together at the same time to kill of life in the Pacific.
BREAKING:
Dead Seals Test Positive For Radiation/Lukaemia
Many
baby seals dying of leukemia-linked disorder along California coast
Blamed
for over 1/3 of recent deaths at San Francisco Bay rescue center
29
August, 2015
Of
the 46 recently weaned northern
elephant seals deaths reported by
the San Francisco Bay Area’s Marine Mammal Center between
April 20 and August 1 of this year, Disseminated Intravascular
Coagulation was listed as a cause of death in 16 — over 1/3 of the
total.
U.S.
National Library of Medicine (emphasis
added):
Disseminated intravascular coagulation (DIC) is a serious disorder in
which the proteins that control blood clotting become over
active… Risk factors for DIC include: Blood transfusion reaction;
Cancer, especially
certain types of leukemia…
DIC “is
a pathological process characterized by the widespread
activation of the clotting cascadethat
results in the formation of blood clots in the small blood vessels
throughout the body… and can ultimately lead to multiple organ
damage… severe bleeding can occur from various sites. DIC
does not occur by itself but
only as a complicating factor from another underlying condition,
usually in those with a critical illness… DIC can lead to
multiorgan failure and widespread bleeding… Causes —
DIC can occur in the following conditions: Solid
tumors and blood cancers (particularly
acute promyelocytic leukemia)…
Sepsis or severe infection… Severe allergic or toxic reactions…
Giant hemangiomas (Kasabach-Merritt syndrome) [and] Large aortic
aneurysms.”
Acute
promyelocytic leukemia is
a subtype of acute myelogenous leukemia (AML), a cancer of the white
blood cells. According to Wikipedia,
“ionizing radiation exposure can increase the risk of AML.
Survivors of the atomic bombings of Hiroshima and Nagasaki had an
increased rate of AML, as did radiologists exposed to high levels of
X-rays”
Journal
of Intensive Care,
2014: Disseminated intravascular coagulation… with enhanced
fibrinolysis is a DIC type usually seen in acute promyelocytic
leukemia (APL)…
The Scientific Standards Committee… defines DIC as ‘an acquired
syndrome [that] can originate from and cause damage to the
microvasculature, which if sufficiently severe, can produce organ
dysfunction’… problems exist with this definition in terms of not
taking into account the type of DIC often seen in acute
leukemias (especially acute promyelocytic leukemia)…
Journal
of Occupational Medicine and Toxicology,
2013: A causal association with leukemia has only been documented to
date for ionizing
radiation,
benzene and treatment with cytostatic drugs… A large number of
studies included in the review referred to the effects of ionizing
radiation, where new
data suggest that the effects of exposure to small doses of ionizing
radiation should probably be reevaluated…
An update of [the Life Span Study (LSS) of A-Bomb survivors] has
shown that exposure
to ionizing radiation at doses as low as
those usually recorded in occupational settings, leukemia incidence
follows a quadratic dose response pattern… Moreover, there
is uncertainty
on whether the proposed safety limits from
the International Commission on Radiological Protection (ICRP) are
appropriate,
since revised LSS data
show that the risk of leukemia remains increased even in groups with
low cumulative exposure to radiation…
Physics
and Radiobiology of Nuclear Medicine (Springer),
Jun 29, 2013: Leukemia
is one of the most common cancers induced by radiation in
humans, accounting for one in five mortalities from
radiocarcinogenesis. Risk of leukemia varies with age, with younger
persons being more prone to radiocarcinogenesis…
Leukemia appears in as early as 2
to 3 years after the exposure,
with an average latent period of 5 to 10 years.
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